Male LH-Independent Sexual Precocity in a 3.5-Year-Old Boy Caused by a Somatic Activating Mutation of the LH Receptor in a Leydig Cell Tumor-Reference-Cited by-同舟云学术

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Male LH-Independent Sexual Precocity in a 3.5-Year-Old Boy Caused by a Somatic Activating Mutation of the LH Receptor in a Leydig Cell Tumor

Published:2002-03 Issue:3 Volume:87 Page:1052-1056
ISSN:0021-972X
Container-title:The Journal of Clinical Endocrinology & Metabolism
language:en
Short-container-title:The Journal of Clinical Endocrinology & Metabolism

Author:

Richter-Unruh A.,Wessels H. T.,Menken U.,Bergmann M.,Schmittmann-Ohters K.,Schaper J.,Tappeser S.,Hauffa B. P.

Publisher

The Endocrine Society

Subject

Biochemistry (medical),Clinical Biochemistry,Endocrinology,Biochemistry,Endocrinology, Diabetes and Metabolism

Cited by 42 articles. 订阅此论文施引文献订阅此论文施引文献，注册后可以免费订阅5篇论文的施引文献，订阅后可以查看论文全部施引文献

1. Central precocious puberty should be taken seriously in children with Leydig cell tumors of the testis after surgical treatment: a tertiary center experience;Asian Journal of Andrology;2024-08-09

2. Revisiting the gonadotropic regulation of mammalian spermatogenesis: evolving lessons during the past decade;Frontiers in Endocrinology;2023-04-14

3. The Adaptive Potential of Nonheritable Somatic Mutations;The American Naturalist;2022-12-01

4. Precocious Puberty in a Boy With Bilateral Leydig Cell Tumors due to a Somatic Gain-of-Function LHCGR Variant;Journal of the Endocrine Society;2022-08-12

5. Sex Cord-Stromal Tumors;Tumors and Tumor-Like Lesions of the Testis and Adjacent Tissues;2022-08

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