RO 31-8220 Activates c-Jun N-Terminal Kinase and Glycogen Synthase in Rat Adipocytes and L6 Myotubes. Comparison to Actions of Insulin*-Reference-Cited by-同舟云学术

RO 31-8220 Activates c-Jun N-Terminal Kinase and Glycogen Synthase in Rat Adipocytes and L6 Myotubes. Comparison to Actions of Insulin*

Published:1999-05-01 Issue:5 Volume:140 Page:2145-2151
ISSN:0013-7227
Container-title:Endocrinology
language:en
Short-container-title:

Author:

Standaert M. L.¹,Bandyopadhyay G.¹,Antwi E. K.¹,Farese R. V.¹

Affiliation:

1. J. A. Haley Veteran’s Hospital Research Service, and Departments of Internal Medicine and Biochemistry/Molecular Biology, University of South Florida College of Medicine, Tampa, Florida 33612

Publisher

The Endocrine Society

Subject

Endocrinology

Link

http://academic.oup.com/endo/article-pdf/140/5/2145/10376426/endo2145.pdf

Reference28 articles.

1. Protein kinase C-ζ as a downstream effector of phosphatidylinositol 3-kinase during insulin stimulation in rat adipocytes. Potential role in glucose transport;Standaert;J Biol Chem,1997

2. Comparative effects of GTPγS and insulin on the activation of Rho, phosphatidylinositol 3-kinase, and protein kinase N in rat adipocytes. Relationship to glucose transport;Standaert;J Biol Chem,1998

3. The selective protein kinase C inhibitor, RO 31-8220, inhibits mitogen-activated protein kinase phosphatase-1 (MKP-1) expression, induces c-Jun expression, and activates Jun N-terminal kinase;Beltman;J Biol Chem,1996

4. Independent human MAP kinase signal transduction pathways defined by MEK and MKK isoforms;Derijard;Science,1995

5. Studies with wortmannin suggest a role for phosphatidylinositol 3-kinase in the activation of glycogen synthase and mitogen-activated protein kinase by insulin in rat adipocytes: Comparison of insulin and protein kinase C modulators;Standaert;Biochem Biophys Res Commun,1995

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