Insulin-Like Growth Factor I Is Essential for Postnatal Growth in Response to Growth Hormone
Author:
Affiliation:
1. Clinical Endocrinology Branch, The National Institute of Diabetes and Digestive and Kidney Diseases, National Institutes of Health, Bethesda, Maryland 20892-1758
Publisher
The Endocrine Society
Subject
Endocrinology
Link
http://academic.oup.com/endo/article-pdf/140/11/5178/10380631/endo5178.pdf
Reference49 articles.
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2. A mammalian model for Laron syndrome produced by targeted disruption of the mouse growth hormone receptor/binding protein gene (the Laron mouse).;Zhou;Proc Natl Acad Sci USA,1997
3. Growth hormone deficiency in ’little’ mice results in aberrant body composition, reduced insulin-like growth factor-I and insulin-like growth factor-binding protein-3 (IGFBP-3), but does not affect IGFBP-2, -1 or -4.;Donahue;J Endocrinol,1993
4. Mice carrying null mutations of the genes encoding insulin-like growth factor I (Igf-1) and type 1 IGF receptor (Igf1r).;Liu;Cell,1993
5. Role of insulin-like growth factors in embryonic and postnatal growth.;Baker;Cell,1993
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