Dysfunction of the Islet Lysosomal System Conveys Impairment of Glucose-Induced Insulin Release in the Diabetic GK Rat*-Reference-Cited by-同舟云学术

Dysfunction of the Islet Lysosomal System Conveys Impairment of Glucose-Induced Insulin Release in the Diabetic GK Rat*

Published:1999-07-01 Issue:7 Volume:140 Page:3045-3053
ISSN:0013-7227
Container-title:Endocrinology
language:en
Short-container-title:

Author:

Salehi Albert¹,Henningsson Ragnar¹,Mosén Henrik¹,Östenson Claes-Göran²,Efendic Suad²,Lundquist Ingmar¹

Affiliation:

1. Department of Pharmacology, University of Lund (A.S., R.H., H.M., I.L.), S-223 62 Lund

2. the Department of Endocrinology, Karolinska Institute and Hospital (C.-G.O., S.E.), S-17176 Stockholm, Sweden

Publisher

The Endocrine Society

Subject

Endocrinology

Link

http://academic.oup.com/endo/article-pdf/140/7/3045/10377653/endo3045.pdf

Reference39 articles.

1. Ca2+, cAMP, and phospholipid-derived messengers in coupling mechanisms of insulin secretion.;Prentki;Physiol Rev,1987

2. Control of insulin secretion: a model involving Ca2+, cAMP and diacylglycerol.;Zawalich;Mol Cell Endocrinol,1990

3. Lysosomal enzyme activities in pancreatic islets from normal and obese hyperglycemic mice.;Lundquist;Metabolism,1985

4. Islet amyloglucosidase activity: some characteristics, and its relation to insulin secretion stimulated by various secretagogues.;Lundquist;Diabetes Res,1986

5. The relationship of islet amyloglucosidase activity and glucose-induced insulin secretion.;Lundquist I. Panagiotidis;Pancreas,1992

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