Osteocyte Protein Expression Is Altered in Low-Turnover Osteoporosis Caused by Mutations in WNT1 and PLS3-Reference-Cited by-同舟云学术

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Osteocyte Protein Expression Is Altered in Low-Turnover Osteoporosis Caused by Mutations in WNT1 and PLS3

Published:2017-04-03 Issue:7 Volume:102 Page:2340-2348
ISSN:0021-972X
Container-title:The Journal of Clinical Endocrinology & Metabolism
language:en
Short-container-title:

Author:

Wesseling-Perry Katherine,Mäkitie Riikka E.,Välimäki Ville-Valtteri,Laine Tero,Laine Christine M.,Välimäki Matti J.,Pereira Renata C.,Mäkitie Outi

Publisher

The Endocrine Society

Subject

Biochemistry (medical),Clinical Biochemistry,Endocrinology,Biochemistry,Endocrinology, Diabetes and Metabolism

Link

http://academic.oup.com/jcem/article-pdf/102/7/2340/18171456/jc.2017-00099.pdf

Reference27 articles.

1. Osteocytes as multifunctional cells;J Musculoskelet Neuronal Interact,2006

2. Osteoporosis and bone mass disorders: from gene pathways to treatments;Trends Endocrinol Metab,2016

3. WNT1 mutations in early-onset osteoporosis and osteogenesis imperfecta;N Engl J Med,2013

4. A novel splice mutation in PLS3 causes X-linked early onset low-turnover osteoporosis;J Bone Miner Res,2015

5. New genetic forms of childhood-onset primary osteoporosis;Horm Res Paediatr,2015

Cited by 23 articles. 订阅此论文施引文献订阅此论文施引文献，注册后可以免费订阅5篇论文的施引文献，订阅后可以查看论文全部施引文献

1. Sclerostin and Wnt Signaling in Idiopathic Juvenile Osteoporosis Using High-Resolution Confocal Microscopy for Three-Dimensional Analyses;Children;2024-07-04

2. Osteoclast-specific Plastin 3 knockout in mice fail to develop osteoporosis despite dramatic increased osteoclast resorption activity;JBMR Plus;2024-01

3. The actin-bundling protein, PLS3, is part of the mechanoresponsive machinery that regulates osteoblast mineralization;Frontiers in Cell and Developmental Biology;2023-11-27

4. Bone fragility and osteoporosis in children and young adults;Journal of Endocrinological Investigation;2023-09-05

5. The intricate mechanism of PLS3 in bone homeostasis and disease;Frontiers in Endocrinology;2023-07-07

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