Inhibition of Dipeptidyl Peptidase-4 Augments Insulin Secretion in Response to Exogenously Administered Glucagon-Like Peptide-1, Glucose-Dependent Insulinotropic Polypeptide, Pituitary Adenylate Cyclase-Activating Polypeptide, and Gastrin-Releasing Peptide in Mice-Reference-Cited by-同舟云学术

Inhibition of Dipeptidyl Peptidase-4 Augments Insulin Secretion in Response to Exogenously Administered Glucagon-Like Peptide-1, Glucose-Dependent Insulinotropic Polypeptide, Pituitary Adenylate Cyclase-Activating Polypeptide, and Gastrin-Releasing Peptide in Mice

Published:2005-04-01 Issue:4 Volume:146 Page:2055-2059
ISSN:0013-7227
Container-title:Endocrinology
language:en
Short-container-title:

Author:

Ahrén Bo¹,Hughes Thomas E.²

Affiliation:

1. Department of Medicine (B.A.), Lund University, SE-221 84 Lund, Sweden

2. Novartis Institutes for Biomedical Research (T.E.H.), Cambridge, Massachusetts 02139

Publisher

The Endocrine Society

Subject

Endocrinology

Link

http://academic.oup.com/endo/article-pdf/146/4/2055/10901060/endo2055.pdf

Reference30 articles.

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3. Glucagon-like peptide 1 (GLP-1): a gut hormone of potential interest in the treatment of diabetes;Ahrén;Bioessays,1998

4. Therapeutic potential of dipeptidyl peptidase IV inhibitors for the treatment of type 2 diabetes;Drucker;Exp Opin Investig Drugs,2003

5. Inhibition of activity of dipeptidyl peptidase IV as a treatment for type 2 diabetes;Holst;Diabetes,1998

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