Chronic Hypersecretion of Luteinizing Hormone in Transgenic Mice Selectively Alters Responsiveness of the -Subunit Gene to Gonadotropin-Releasing Hormone and Estrogens
Author:
Publisher
The Endocrine Society
Subject
Endocrinology,Molecular Biology,General Medicine
Cited by 13 articles. 订阅此论文施引文献 订阅此论文施引文献,注册后可以免费订阅5篇论文的施引文献,订阅后可以查看论文全部施引文献
1. Animal models for aberrations of gonadotropin action;Reviews in Endocrine and Metabolic Disorders;2011-04-12
2. Origin and Identity of Adrenocortical Tumors in Inhibin Knockout Mice: Implications for Cellular Plasticity in the Adrenal Cortex;Molecular Endocrinology;2006-11-01
3. Early Multipotential Pituitary Focal Hyperplasia in the α-Subunit of Glycoprotein Hormone-Driven Pituitary Tumor-Transforming Gene Transgenic Mice;Molecular Endocrinology;2005-05
4. Multiple sites of tumorigenesis in transgenic mice overproducing hCG;Molecular and Cellular Endocrinology;2005-04
5. Targeted overexpression of calcitonin in gonadotrophs of transgenic mice leads to chronic hypoprolactinemia;Molecular and Cellular Endocrinology;2005-01
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