SAP97-mediated rescue of NMDA receptor surface distribution in a neuronal model of Huntington's disease
Author:
Affiliation:
1. Department of Physiology and Centre for Brain Research; The University of Auckland; Auckland New Zealand
Funder
University of Auckland
Auckland Medical Research Foundation
Publisher
Wiley
Subject
Cognitive Neuroscience
Link
http://onlinelibrary.wiley.com/wol1/doi/10.1002/hipo.22995/fullpdf
Reference95 articles.
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3. Altered cortical glutamate receptor function in the R6/2 model of Huntington's disease;Andre;Journal of Neurophysiology,2006
4. Shank3 Is part of a zinc-sensitive signaling system that regulates excitatory synaptic strength;Arons;Journal of Neuroscience,2016
5. Autism-associated mutations in ProSAP2/Shank3 impair synaptic transmission and neurexin-neuroligin-mediated transsynaptic signaling;Arons;Journal of Neuroscience,2012
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3. Piperine mitigates behavioral impairments and provides neuroprotection against 3-nitropropinoic acid-induced Huntington disease-like symptoms;Nutritional Neuroscience;2020-02-25
4. Cell-Autonomous and Non-cell-Autonomous Pathogenic Mechanisms in Huntington’s Disease: Insights from In Vitro and In Vivo Models;Neurotherapeutics;2019-09-16
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