Epileptiform activity in the CA1 region of the hippocampus becomes refractory to attenuation by cannabinoids in part because of endogenous γ-aminobutyric acid type B receptor activity
Author:
Publisher
Wiley
Subject
Cellular and Molecular Neuroscience
Link
http://onlinelibrary.wiley.com/wol1/doi/10.1002/jnr.23027/fullpdf
Reference41 articles.
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4. Transient suppression of GABAA-receptor-mediated IPSPs after epileptiform burst discharges in CA1 pyramidal cells;Beau;J Neurophysiol,1998
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1. Effect of synthetic cannabinoids on spontaneous neuronal activity: Evaluation using Ca 2+ spiking and multi-electrode arrays;European Journal of Pharmacology;2016-09
2. Possible involvement of the CA1 GABAA receptors upon acquisition and expression of the ACPA-induced place preference in mice;Physiology & Behavior;2016-07
3. Endocannabinoids and epilepsy;Cannabinoids in Neurologic and Mental Disease;2015
4. The Role of CB1-Receptors in the Proconvulsant Effect of Leptin on Penicillin-Induced Epileptiform Activity in Rats;CNS Neuroscience & Therapeutics;2013-03-25
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