Galectin-1 promotes an M2 macrophage response to polydioxanone scaffolds

Author:

Abebayehu Daniel12,Spence Andrew1,Boyan Barbara D.23,Schwartz Zvi24,Ryan John J.1,McClure Michael J.25

Affiliation:

1. Department of Biology; Virginia Commonwealth University; Richmond Virginia

2. Department of Biomedical Engineering, School of Engineering; Virginia Commonwealth University; Richmond Virginia

3. Wallace H. Coulter Department of Biomedical Engineering; Georgia Institute of Technology; Atlanta Georgia

4. Department of Periodontics; University of Texas Health Sciences Center at San Antonio; San Antonio Texas

5. Physical Medicine and Rehabilitation Service; Hunter Holmes McGuire VA Medical Center; Richmond Virginia

Funder

Department of Veterans Affairs Interprofessional Polytrauma and Traumatic Brain Injury Rehabilitation Research Fellowship

National Institutes for Health

Publisher

Wiley

Subject

Metals and Alloys,Biomedical Engineering,Biomaterials,Ceramics and Composites

Reference51 articles.

1. Macrophages and angiogenesis;Sunderkotter;J Leukoc Biol,1994

2. Galectin-1 induces 12/15-lipoxygenase expression in murine macrophages and favors their conversion toward a pro-resolving phenotype;Rostoker;Prostaglandins Other Lipid Mediat,2013

3. MAP kinase activation in macrophages;Rao;J Leukoc Biol,2001

4. Altered macrophage phenotype transition impairs skeletal muscle regeneration;Wang;Am J Pathol,2014

5. The role of fibrosis in Duchenne muscular dystrophy;Klingler;Acta Myol,2012

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