Normal development of spinal axons in early embryo stages and posterior locomotor function is independent of GAL-1
Author:
Affiliation:
1. Departamento de Química Biológica; Instituto de Química y Físico Química Biológica, Universidad de Buenos Aires; Buenos Aires Argentina
2. Laboratory of Molecular Neurobiology; BIOMED UCA-CONICET; Buenos Aires Argentina
Funder
CONICET
Foundation Florencio Fiorini
Publisher
Wiley
Subject
General Neuroscience
Link
http://onlinelibrary.wiley.com/wol1/doi/10.1002/cne.24243/fullpdf
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5. Analysis of the L1-deficient mouse phenotype reveals cross-talk between Sema3A and L1 signaling pathways in axonal guidance;Castellani;Neuron,2000
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