METTL3 is aberrantly expressed in endometriosis and suppresses proliferation, invasion, and migration of endometrial stromal cells
Author:
Affiliation:
1. Department of Obstetrics and Gynecology, Union Hospital, Tongji Medical College Huazhong University of Science and Technology Wuhan China
Publisher
Wiley
Subject
General Medicine
Link
https://onlinelibrary.wiley.com/doi/pdf/10.1002/kjm2.12626
Reference32 articles.
1. Rethinking mechanisms, diagnosis and management of endometriosis
2. Endometriosis
3. Endometriosis—It is not just benign
4. Endoglin promotes cell migration and invasion in endometriosis by regulating EMT
5. The motile and invasive capacity of human endometrial stromal cells: implications for normal and impaired reproductive function
Cited by 3 articles. 订阅此论文施引文献 订阅此论文施引文献，注册后可以免费订阅5篇论文的施引文献，订阅后可以查看论文全部施引文献
1. m6A methylation of RNF43 inhibits the progression of endometriosis through regulating oxidative phosphorylation via NDUFS1;Journal of Cellular Physiology;2024-07-10
2. METTL3-regulated m6A modification impairs the decidualization of endometrial stromal cells by regulating YTHDF2-mediated degradation of FOXO1 mRNA in endometriosis-related infertility;Reproductive Biology and Endocrinology;2023-10-27
3. The Expression of TGF-β1, SMAD3, ILK and miRNA-21 in the Ectopic and Eutopic Endometrium of Women with Endometriosis;International Journal of Molecular Sciences;2023-01-26
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