Liver-specific knockout of GRP94 in mice disrupts cell adhesion, activates liver progenitor cells, and accelerates liver tumorigenesis-Reference-Cited by-同舟云学术

Liver-specific knockout of GRP94 in mice disrupts cell adhesion, activates liver progenitor cells, and accelerates liver tumorigenesis

Published:2014-01-27 Issue:3 Volume:59 Page:947-957
ISSN:0270-9139
Container-title:Hepatology
language:en
Short-container-title:Hepatology

Author:

Chen Wan-Ting¹,Tseng Chun-Chih¹,Pfaffenbach Kyle¹,Kanel Gary²,Luo Biquan¹,Stiles Bangyan L.³,Lee Amy S.¹

Affiliation:

1. Department of Biochemistry and Molecular Biology; University of Southern California, Keck School of Medicine, USC Norris Comprehensive Cancer Center; Los Angeles CA

2. Department of Pathology; University of Southern California, Keck School of Medicine; Los Angeles CA

3. Department of Pharmacology and Pharmaceutical Sciences; University of Southern California, Keck School of Medicine; Los Angeles CA

Publisher

Wiley

Subject

Hepatology

Link

http://onlinelibrary.wiley.com/wol1/doi/10.1002/hep.26711/fullpdf

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4. GRP94 in ER quality control and stress responses;Eletto;Semin Cell Dev Biol,2010

5. Biology and pathobiology of gap junctional channels in hepatocytes;Vinken;Hepatology,2008

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