Endoplasmic reticulum stress induces hepatic steatosis via increased expression of the hepatic very low‐density lipoprotein receptor-Reference-Cited by-同舟云学术

Endoplasmic reticulum stress induces hepatic steatosis via increased expression of the hepatic very low‐density lipoprotein receptor

Published:2013-04 Issue:4 Volume:57 Page:1366-1377
ISSN:0270-9139
Container-title:Hepatology
language:en
Short-container-title:Hepatology

Author:

Jo Hyunsun¹,Choe Sung Sik¹,Shin Kyung Cheul¹,Jang Hagoon¹,Lee Jae Ho¹,Seong Je Kyung²,Back Sung Hoon³,Kim Jae Bum¹⁴

Affiliation:

1. School of Biological Sciences, Institute of Molecular Biology and Genetics, University of Ulsan, Ulsan, Korea

2. Department of Anatomy and Cell Biology, College of Veterinary Medicine and Research Institute for Veterinary Science, University of Ulsan, Ulsan, Korea

3. School of Biological Sciences, World Class University, University of Ulsan, Ulsan, Korea

4. Department of Biophysics and Chemical Biology, Seoul National University, Seoul, Korea

Funder

National Creative Research Initiative

World Class University Program

Ministry of Education, Science, and Technology

Publisher

Wiley

Subject

Hepatology

Link

https://onlinelibrary.wiley.com/doi/pdf/10.1002/hep.26126

Reference49 articles.

1. Signal integration in the endoplasmic reticulum unfolded protein response

2. The Unfolded Protein Response: From Stress Pathway to Homeostatic Regulation

3. Endoplasmic Reticulum Stress in Disease Pathogenesis

4. The Role for Endoplasmic Reticulum Stress in Diabetes Mellitus

5. The Role of Endoplasmic Reticulum Stress in the Progression of Atherosclerosis

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