Iron overloaded polarizes macrophage to proinflammation phenotype through ROS/acetyl-p53 pathway-Reference-Cited by-同舟云学术

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Iron overloaded polarizes macrophage to proinflammation phenotype through ROS/acetyl-p53 pathway

Published:2018-07-10 Issue:8 Volume:7 Page:4012-4022
ISSN:2045-7634
Container-title:Cancer Medicine
language:en
Short-container-title:Cancer Med

Author:

Zhou Yun¹^ORCID,Que Ke-Ting¹,Zhang Zhen¹,Yi Zu J.¹,Zhao Ping X.¹,You Yu¹,Gong Jian-Ping¹,Liu Zuo-Jin¹

Affiliation:

1. Chongqing Medical University; Chongqing China

Funder

National Natural Science Foundation of China

Talent Project of The Second Affiliated Hospital of Chongqing Medical University

Publisher

Wiley

Subject

Cancer Research,Radiology Nuclear Medicine and imaging,Oncology

Link

http://onlinelibrary.wiley.com/wol1/doi/10.1002/cam4.1670/fullpdf

Reference25 articles.

1. Macrophage iron homeostasis and polarization in the context of cancer;Jung;Immunobiology,2015

2. The role of iron metabolism as a mediator of macrophage inflammation and lipid handling in atherosclerosis;Habib;Front Pharmacol,2014

3. ROS and p53: a versatile partnership;Liu;Free Radic Biol Med,2008

4. Beyond oxidative stress: an immunologist's guide to reactive oxygen species;Nathan;Nat Rev Immunol,2013

5. ROS inhibitor N-acetyl-L-cysteine antagonizes the activity of proteasome inhibitors;Halasi;Biochem J,2013

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