BAFF overexpression and accelerated glomerular disease in mice with an incomplete genetic predisposition to systemic lupus erythematosus-Reference-Cited by-同舟云学术

BAFF overexpression and accelerated glomerular disease in mice with an incomplete genetic predisposition to systemic lupus erythematosus

Published:2005 Issue:7 Volume:52 Page:2080-2091
ISSN:0004-3591
Container-title:Arthritis & Rheumatism
language:en
Short-container-title:Arthritis Rheum

Author:

Stohl William,Xu Dong,Kim Kyoung Soo,Koss Michael N.,Jorgensen Trine N.,Deocharan Bisram,Metzger Troy E.,Bixler Sarah A.,Hong Yeon Sik,Ambrose Christine M.,Mackay Fabienne,Morel Laurence,Putterman Chaim,Kotzin Brian L.,Kalled Susan L.

Publisher

Wiley

Subject

Pharmacology (medical),Immunology,Rheumatology,Immunology and Allergy

Reference58 articles.

1. BAFF, a Novel Ligand of the Tumor Necrosis Factor Family, Stimulates B Cell Growth

2. BLyS: Member of the Tumor Necrosis Factor Family and B Lymphocyte Stimulator

3. TALL-1 is a novel member of the TNF family that is down-regulated by mitogens

4. Identification and Characterization of a Novel Cytokine, THANK, aTNF Homologue That ActivatesApoptosis, Nuclear Factor-κB, and c-Jun NH2-Terminal Kinase

5. Characterization of a New Member of the TNF Family Expressed on Antigen Presenting Cells

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