Oncogenic effects of exosomes in γ‐herpesvirus‐associated neoplasms
Author:
Affiliation:
1. Microbiology and Immunology Department, Basic Medical School Guangdong Medical University Dongguan Guangdong China
2. The Second School of Clinical Medicine Guangdong Medical University Dongguan Guangdong China
Funder
Natural Science Foundation of Guangdong Province
Publisher
Wiley
Subject
Cell Biology,Clinical Biochemistry,Physiology
Link
https://onlinelibrary.wiley.com/doi/pdf/10.1002/jcp.28573
Reference82 articles.
1. Kaposi's Sarcoma-Associated Herpesvirus MicroRNAs Target IRAK1 and MYD88, Two Components of the Toll-Like Receptor/Interleukin-1R Signaling Cascade, To Reduce Inflammatory-Cytokine Expression
2. Regulation of Tumor Necrosis Factor-Like Weak Inducer of Apoptosis Receptor Protein (TWEAKR) Expression by Kaposi's Sarcoma-Associated Herpesvirus MicroRNA Prevents TWEAK-Induced Apoptosis and Inflammatory Cytokine Expression
3. Exosomal HIF1α supports invasive potential of nasopharyngeal carcinoma-associated LMP1-positive exosomes
4. The labyrinth of interactions of Epstein-Barr virus-encoded small RNAs
5. Epstein–Barr virus-infected cells release Fas ligand in exosomal fractions and induce apoptosis in recipient cells via the extrinsic pathway
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