Neurochemical correlates of caudate atrophy in Huntington's disease-Reference-Cited by-同舟云学术

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Neurochemical correlates of caudate atrophy in Huntington's disease

Published:2014-01-17 Issue:3 Volume:29 Page:327-335
ISSN:0885-3185
Container-title:Movement Disorders
language:en
Short-container-title:Mov Disord.

Author:

Padowski Jeannie M.¹²,Weaver Kurt E.¹²,Richards Todd L.¹²,Laurino Mercy Y.³,Samii Ali⁴,Aylward Elizabeth H.¹²,Conley Kevin E.¹²

Affiliation:

1. Department of Radiology; University of Washington School of Medicine; Seattle Washington USA

2. Integrated Brain Imaging Center; University of Washington School of Medicine; Seattle Washington USA

3. Department of Medical Genetics; University of Washington School of Medicine; Seattle Washington USA

4. Department of Neurology; University of Washington School of Medicine; Seattle Washington USA

Publisher

Wiley

Subject

Neurology (clinical),Neurology

Link

http://onlinelibrary.wiley.com/wol1/doi/10.1002/mds.25801/fullpdf

Reference47 articles.

1. [No authors listed] A novel gene containing a trinucleotide repeat that is expanded and unstable on Huntington's disease chromosomes. The Huntington's Disease Collaborative Research Group;Cell,1993

2. The association of CAG repeat length with clinical progression in Huntington disease;Rosenblatt;Neurology,2006

3. Longitudinal change in regional brain volumes in prodromal Huntington disease;Aylward;J Neurol Neurosurg Psychiatry,2011

4. The search for sensitive biomarkers in presymptomatic Huntington disease;Henry;J Cereb Blood Flow Metab,2012

5. Change in MRI striatal volumes as a biomarker in preclinical Huntington's disease;Aylward;Brain Res Bull,2007

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