A critical role for TRAIL in resolution of granulomatous experimental autoimmune thyroiditis-Reference-Cited by-同舟云学术

A critical role for TRAIL in resolution of granulomatous experimental autoimmune thyroiditis

Published:2008-09-22 Issue:4 Volume:216 Page:505-513
ISSN:0022-3417
Container-title:The Journal of Pathology
language:en
Short-container-title:J. Pathol.

Author:

Fang Y¹²,Sharp GC²³,Yagita H⁴,Braley‐Mullen H¹²⁵

Affiliation:

1. Research Service, Harry S Truman Memorial VA Hospital, University of Missouri, Columbia, MO 65212, USA

2. Department of Internal Medicine, School of Medicine, University of Missouri, Columbia, MO 65212, USA

3. Department of Pathology, School of Medicine, University of Missouri, Columbia, MO 65212, USA

4. Department of Immunology, Juntendo University School of Medicine, Tokyo, Japan

5. Department of Molecular Microbiology and Immunology, School of Medicine, University of Missouri, Columbia, MO 65212, USA

Publisher

Wiley

Subject

Pathology and Forensic Medicine

Link

https://onlinelibrary.wiley.com/doi/pdf/10.1002/path.2428

Reference38 articles.

1. Induction of experimental autoimmune thyroiditis in mice with in vitro activated splenic T cells

2. Induction of severe granulomatous experimental autoimmune thyroiditis in mice by effector cells activated in the presence of anti-interleukin 2 receptor antibody.

3. Interleukin‐12 promotes activation of effector cells that induce a severe destructive granulomatous form of murine experimental autoimmune thyroiditis;Braley‐Mullen H;Am J Pathol,1998

4. Adoptive Transfer Murine Model of Granulomatous Experimental Autoimmune Thyroiditis

5. Regulation of the Induction and Resolution of Granulomatous Experimental Autoimmune Thyroiditis in Mice by CD8+ T Cells

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