Genomewide Comprehensive Analysis Reveals Critical Cooperation Between Smad and c‐Fos in RANKL‐Induced Osteoclastogenesis-Reference-Cited by-同舟云学术

Genomewide Comprehensive Analysis Reveals Critical Cooperation Between Smad and c‐Fos in RANKL‐Induced Osteoclastogenesis

Published:2015-04-16 Issue:5 Volume:30 Page:869-877
ISSN:0884-0431
Container-title:Journal of Bone and Mineral Research
language:en
Short-container-title:J Bone Miner Res

Author:

Omata Yasunori¹,Yasui Tetsuro¹,Hirose Jun¹,Izawa Naohiro¹,Imai Yuuki²,Matsumoto Takumi¹,Masuda Hironari¹,Tokuyama Naoto¹,Nakamura Shinya¹,Tsutsumi Shuichi³,Yasuda Hisataka⁴,Okamoto Kazuo⁵,Takayanagi Hiroshi⁵,Hikita Atsuhiko⁶,Imamura Takeshi⁶,Matsuo Koichi⁷,Saito Taku¹,Kadono Yuho¹,Aburatani Hiroyuki³,Tanaka Sakae¹

Affiliation:

1. Department of Orthopaedic SurgeryFaculty of MedicineThe University of TokyoTokyoJapan

2. Division of Integrative PathophysiologyProteo‐Science CenterGraduate School of Medicine, Ehime UniversityEhimeJapan

3. Genome Science DivisionResearch Center for Advanced Science and Technology (RCAST)The University of TokyoTokyoJapan

4. Bioindustry DivisionOriental Yeast Co. Ltd.TokyoJapan

5. Department of Immunology, Graduate School of Medicine and Faculty of MedicineThe University of Tokyo, Tokyo, Japan; Japan Science and Technology Agency (JST), Exploratory Research for Advanced Technology (ERATO) Program, Takayanagi Osteonetwork ProjectTokyoJapan

6. Department of Molecular Medicine for PathogenesisGraduate School of MedicineEhime UniversityEhimeJapan

7. Laboratory of Cell and Tissue BiologySchool of MedicineKeio UniversityTokyoJapan

Publisher

Wiley

Subject

Orthopedics and Sports Medicine,Endocrinology, Diabetes and Metabolism

Link

https://onlinelibrary.wiley.com/doi/pdf/10.1002/jbmr.2418

Reference37 articles.

1. Signaling Axis in Osteoclast Biology and Therapeutic Targeting in the RANKL/RANK/OPG System

2. Osteoprotegerin Ligand Is a Cytokine that Regulates Osteoclast Differentiation and Activation

3. Modulation of Osteoclast Differentiation and Function by the New Members of the Tumor Necrosis Factor Receptor and Ligand Families