Reversion of levodopa-induced motor fluctuations by the A2A antagonist CSC is associated with an increase in striatal preprodynorphin mRNA expression in 6-OHDA-lesioned rats
Author:
Publisher
Wiley
Subject
Cellular and Molecular Neuroscience
Reference65 articles.
1. Striatal fosB Expression Is Causally Linked with l-DOPA-Induced Abnormal Involuntary Movements and the Associated Upregulation of Striatal Prodynorphin mRNA in a Rat Model of Parkinson's Disease
2. Time course of striatal ΔFosB-like immunoreactivity and prodynorphin mRNA levels after discontinuation of chronic dopaminomimetic treatment
3. Agonist-Induced Phosphorylation of the κ-Opioid Receptor
4. Adenosine A2Aantagonism reverses levodopa-induced motor alterations in hemiparkinsonian rats
5. Differential regulation of GAD67, enkephalin and dynorphin mRNAs by chronic-intermittentL-dopa and A2A receptor blockade plusL-Dopa in dopamine-denervated rats
Cited by 14 articles. 订阅此论文施引文献 订阅此论文施引文献,注册后可以免费订阅5篇论文的施引文献,订阅后可以查看论文全部施引文献
1. Role of adenosine A2A receptors in motor control: relevance to Parkinson’s disease and dyskinesia;Journal of Neural Transmission;2018-02-02
2. Adenosine A2A Receptor Antagonists as Drugs for Symptomatic Control of Parkinson’s Disease in Preclinical Studies;Current Topics in Neurotoxicity;2015
3. A Critical Evaluation of Behavioral Rodent Models of Motor Impairment Used for Screening of Antiparkinsonian Activity: The Case of Adenosine A2A Receptor Antagonists;Neurotoxicity Research;2013-12-10
4. Symptomatic and Neuroprotective Effects of A2A Receptor Antagonists in Parkinson’s Disease;Adenosine;2012-07-23
5. Neurotoxin-based models of Parkinson's disease;Neuroscience;2012-06
1.学者识别学者识别
2.学术分析学术分析
3.人才评估人才评估
"同舟云学术"是以全球学者为主线,采集、加工和组织学术论文而形成的新型学术文献查询和分析系统,可以对全球学者进行文献检索和人才价值评估。用户可以通过关注某些学科领域的顶尖人物而持续追踪该领域的学科进展和研究前沿。经过近期的数据扩容,当前同舟云学术共收录了国内外主流学术期刊6万余种,收集的期刊论文及会议论文总量共计约1.5亿篇,并以每天添加12000余篇中外论文的速度递增。我们也可以为用户提供个性化、定制化的学者数据。欢迎来电咨询!咨询电话:010-8811{复制后删除}0370
www.globalauthorid.com
TOP
Copyright © 2019-2024 北京同舟云网络信息技术有限公司 京公网安备11010802033243号 京ICP备18003416号-3