Trisomy 12 in HESC leads to no selective in vivo growth advantage in teratomas, but induces an increased abundance of renal development-Reference-Cited by-同舟云学术

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Trisomy 12 in HESC leads to no selective in vivo growth advantage in teratomas, but induces an increased abundance of renal development

Published:2007 Issue:6 Volume:100 Page:1518-1525
ISSN:0730-2312
Container-title:Journal of Cellular Biochemistry
language:en
Short-container-title:J. Cell. Biochem.

Author:

Gertow Karin,Cedervall Jessica,Unger Christian,Szöke Krisztina,Blennow Elisabeth,Imreh Marta P.,Ährlund-Richter Lars

Publisher

Wiley

Subject

Cell Biology,Molecular Biology,Biochemistry

Reference26 articles.

1. Karyotypic Stability, Genotyping, Differentiation, Feeder-Free Maintenance, and Gene Expression Sampling in Three Human Embryonic Stem Cell Lines Derived Prior to August 9, 2001

2. Karyotype of human ES cells during extended culture

3. Properties of four human embryonic stem cell lines maintained in a feeder-free culture system

4. Overexpression of NANOG in human ES cells enables feeder-free growth while inducing primitive ectoderm features

5. Culture and Characterization of Human Embryonic Stem Cells

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1. Trisomy 12 compromises the mesendodermal differentiation propensity of human pluripotent stem cells;In Vitro Cellular & Developmental Biology - Animal;2024-01-02

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3. Genetic and epigenetic factors which modulate differentiation propensity in human pluripotent stem cells;Human Reproduction Update;2018-01-25

4. Musculoskeletal Tissue Regeneration: the Role of the Stem Cells;Regenerative Engineering and Translational Medicine;2017-08-29

5. The tumorigenic potential of pluripotent stem cells: What can we do to minimize it?;BioEssays;2016-07

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