Paclitaxel inhibits osteoclast formation and bone resorption via influencing mitotic cell cycle arrest and RANKL-induced activation of NF-κB and ERK-Reference-Cited by-同舟云学术

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Paclitaxel inhibits osteoclast formation and bone resorption via influencing mitotic cell cycle arrest and RANKL-induced activation of NF-κB and ERK

Published:2012-01-20 Issue:3 Volume:113 Page:946-955
ISSN:0730-2312
Container-title:Journal of Cellular Biochemistry
language:en
Short-container-title:J. Cell. Biochem.

Author:

Ang Estabelle S. M.,Pavlos Nathan J.,Chim Shek Man,Feng Hao Tian,Scaife Robin M.,Steer James H.,Zheng Ming H.,Xu Jiake

Publisher

Wiley

Subject

Cell Biology,Molecular Biology,Biochemistry

Link

http://onlinelibrary.wiley.com/wol1/doi/10.1002/jcb.23423/fullpdf

Reference33 articles.

1. Taxanes: Microtubule and centrosome targets, and cell cycle dependent mechanisms of action;Abal;Curr Cancer Drug Targets,2003

2. Mangiferin attenuates osteoclastogenesis, bone resorption, and RANKL-induced activation of NF-kappaB and ERK;Ang;J Cell Biochem,2011a

3. Naringin abrogates osteoclastogenesis and bone resorption via the inhibition of RANKL-induced NF-kappaB and ERK activation;Ang;FEBS Lett,2011b

4. Calcium/calmodulin-dependent kinase activity is required for efficient induction of osteoclast differentiation and bone resorption by receptor activator of nuclear factor kappa B ligand (RANKL);Ang;J Cell Physiol,2007

5. Taxol-induced apoptosis depends on MAP kinase pathways (ERK and p38) and is independent of p53;Bacus;Oncogene,2001

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