The functional consequences of sodium channel NaV 1.8 in human left ventricular hypertrophy

Author:

Ahmad Shakil12,Tirilomis Petros2,Pabel Steffen1,Dybkova Nataliya2,Hartmann Nico2,Molina Cristina E.3,Tirilomis Theodoros4,Kutschka Ingo4,Frey Norbert5,Maier Lars S.1,Hasenfuss Gerd2,Streckfuss-Bömeke Katrin2,Sossalla Samuel12

Affiliation:

1. Department of Internal Medicine II; University Medical Center Regensburg; Regensburg Germany

2. Department of Cardiology and Pneumology; University Hospital, Georg-August University Goettingen, and DZHK (German Centre for Cardiovascular Research), partner site Goettingen; Goettingen Germany

3. Institute of Experimental Cardiovascular Research; University Medical Center Hamburg-Eppendorf; Hamburg Germany

4. Department of Thoracic, Cardiac and Vascular Surgery; University Hospital, Georg-August University Goettingen; Goettingen Germany

5. Department of Internal Medicine III, Molecular Cardiology and Angiology; University Medical Center, Schleswig-Holstein, Campus Kiel; Kiel Germany

Funder

Deutsche Forschungsgemeinschaft

ReForM-Programme

Bundesministerium für Bildung und Forschung

Marga und Walter Boll-Stiftung

Publisher

Wiley

Subject

Cardiology and Cardiovascular Medicine

Reference40 articles.

1. The spectrum of left ventricular hypertrophy in a general population sample: the Framingham Study;Savage;Circulation,1987

2. Regression of left ventricular hypertrophy; what are appropriate therapeutic objectives?;Sheridan;Br J Clin Pharmacol,1999

3. Prevalence and natural history of electrocardiographic left ventricular hypertrophy;Kannel;Am J Med,1983

4. Prognostic implications of echocardiographically determined left ventricular mass in the Framingham Heart Study;Levy;N Engl J Med,1990

5. 2016 ESC guidelines for the diagnosis and treatment of acute and chronic heart failure;Ponikowski;Rev Esp Cardiol (Engl Ed),2016

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