An inhibitory mechanism of action of coiled‐coil peptides against type three secretion system from enteropathogenicEscherichia coli-Reference-Cited by-同舟云学术

An inhibitory mechanism of action of coiled‐coil peptides against type three secretion system from enteropathogenicEscherichia coli

Published:2019-02-11 Issue:3 Volume:25 Page:
ISSN:1075-2617
Container-title:Journal of Peptide Science
language:en
Short-container-title:J Pep Sci

Author:

Larzábal Mariano¹,Baldoni Hector A.²³,Suvire Fernando D.²⁴,Curto Lucrecia M.⁵⁶,Gomez Gabriela E.⁵⁶,Da Silva Wanderson Marques¹,Giudicessi Silvana L.⁷⁸,Camperi Silvia A.⁷⁸,Delfino Jose M.⁵⁶,Cataldi Angel A.¹,Enriz Daniel²⁴

Affiliation:

1. Instituto IABIMO INTA‐CONICET Castelar Buenos Aires Argentina

2. Facultad de Química, Bioquímica y FarmaciaUniversidad Nacional de San Luis San Luis Argentina

3. IMASL‐CONICET San Luis Argentina

4. IMIBIO‐CONICET San Luis Argentina

5. Facultad de Farmacia y Bioquímica, Departamento de Química BiológicaUniversidad de Buenos Aires (UBA) Buenos Aires Argentina

6. Universidad de Buenos Aires‐CONICET, Instituto de Química y Fisicoquímica Biológicas (IQUIFIB) Buenos Aires Argentina

7. Facultad de Farmacia y Bioquímica, Cátedra de BiotecnologíaUniversidad de Buenos Aires (UBA) Buenos Aires Argentina

8. CONICET—Universidad de Buenos Aires, Instituto de Nanobiotecnología (NANOBIOTEC) Buenos Aires Argentina

Funder

Academy of Motion Picture Arts and Sciences

Pipeline and Hazardous Materials Safety Administration

Secretaría de Ciencia y Técnica, Universidad de Buenos Aires

Consejo Nacional de Investigaciones Científicas y Técnicas

Universidad de Buenos Aires

Universidad Nacional de San Luis

Publisher

Wiley

Subject

Organic Chemistry,Drug Discovery,Pharmacology,Molecular Biology,Molecular Medicine,General Medicine,Biochemistry,Structural Biology

Link

https://onlinelibrary.wiley.com/doi/pdf/10.1002/psc.3149

Reference53 articles.

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4. Shiga toxin-associated hemolytic uremic syndrome: absence of recurrence after renal transplantation

5. A genetic locus of enterocyte effacement conserved among diverse enterobacterial pathogens.

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