Deletion of the Antiphospholipid Syndrome Autoantigen β2-Glycoprotein I Potentiates the Lupus Autoimmune Phenotype in a Toll-like Receptor 7-Mediated Murine Model-Reference-Cited by-同舟云学术

Deletion of the Antiphospholipid Syndrome Autoantigen β2-Glycoprotein I Potentiates the Lupus Autoimmune Phenotype in a Toll-like Receptor 7-Mediated Murine Model

Published:2014-07-28 Issue:8 Volume:66 Page:2270-2280
ISSN:2326-5191
Container-title:Arthritis & Rheumatology
language:en
Short-container-title:Arthritis & Rheumatology

Author:

Giannakopoulos Bill¹,Mirarabshahi Peyman¹,Qi Miao¹,Weatherall Chris¹,Qi Jian Cheng¹,Tanaka Kumiko¹,Millar Ewan²,Vonthethoff Leon¹,Gatto Dominique³,Spielman Derek⁴,Krilis Steven A.¹

Affiliation:

1. St. George Hospital and University of New South Wales; Kogarah, New South Wales Australia

2. St. George Hospital, Kogarah, New South Wales, Australia, Kinghorn Cancer Centre and Garvan Institute of Medical Research, Darlinghurst, New South Wales, Australia, University of Western Sydney, Campbelltown Campus, Campbelltown, New South Wales, Australia, and University of New South Wales; Kensington, New South Wales Australia

3. Garvan Institute of Medical Research and St. Vincent's Clinical School, University of New South Wales; Darlinghurst, New South Wales Australia

4. University of Sydney, Camperdown Campus; Camperdown, New South Wales Australia

Funder

National Health and Medical Research Council

Publisher

Wiley

Subject

Immunology,Rheumatology,Immunology and Allergy

Reference39 articles.

1. International consensus statement on an update of the classification criteria for definite antiphospholipid syndrome (APS);Miyakis;J Thromb Haemost,2006

2. The pathogenesis of the antiphospholipid syndrome;Giannakopoulos;N Engl J Med,2013

3. Anti-phospholipid antibodies are directed against a complex antigen that includes a lipid-binding inhibitor of coagulation: β2-glycoprotein I (apolipoprotein H);McNeil;Proc Natl Acad Sci U S A,1990