Depletion of alveolar macrophages in CD11c diphtheria toxin receptor mice produces an inflammatory response-Reference-Cited by-同舟云学术

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Depletion of alveolar macrophages in CD11c diphtheria toxin receptor mice produces an inflammatory response

Published:2015-03-23 Issue:2 Volume:3 Page:71-81
ISSN:2050-4527
Container-title:Immunity, Inflammation and Disease
language:en
Short-container-title:

Author:

Roberts Lydia M.¹,Ledvina Hannah E.¹,Tuladhar Shraddha¹,Rana Deepa¹,Steele Shaun P.²,Sempowski Gregory D.³,Frelinger Jeffrey A.¹

Affiliation:

1. Department of Immunobiology; University of Arizona; Tucson Arizona USA

2. Department of Microbiology and Immunology; University of North Carolina at Chapel Hill; Chapel Hill North Carolina USA

3. Duke Human Vaccine Institute; Durham North Carolina USA

Funder

National Institutes of Health

Publisher

Wiley

Subject

Immunology,Immunology and Allergy

Reference33 articles.

1. Identification of early interactions between Francisella and the host;Roberts;Infect. Immun.,2014

2. Decreased intrapulmonary killing of Mycoplasma pulmonis after short-term exposure to NO2 is associated with damaged alveolar macrophages;Davis;Am. Rev. Respir. Dis.,1992

3. Macrophage defences against respiratory tract infections;Gordon;Br. Med. Bull.,2002

4. Depletion of alveolar macrophages exerts protective effects in pulmonary tuberculosis in mice;Leemans;J. Immunol.,2001

5. Interaction between the legionnaires’ disease bacterium (Legionella pneumophila) and human alveolar macrophages. Influence of antibody, lymphokines, and hydrocortisone;Nash;J. Clin. Invest.,1984

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