Interactions between superoxide dismutase and paraoxonase polymorphic variants in nonsyndromic cleft lip with or without cleft palate in the Brazilian population-Reference-Cited by-同舟云学术

Interactions between superoxide dismutase and paraoxonase polymorphic variants in nonsyndromic cleft lip with or without cleft palate in the Brazilian population

Published:2018-09-21 Issue:2 Volume:60 Page:185-196
ISSN:0893-6692
Container-title:Environmental and Molecular Mutagenesis
language:en
Short-container-title:Environ Mol Mutagen

Author:

Machado Renato Assis¹^ORCID,Oliveira Silva Carolina¹,Persuhn Darlene Camati²,Melo Cavalcanti Dantas Vanessa²,Almeida Reis Silvia Regina³,Wu Tao⁴,Line Sergio Roberto⁵,Martelli‐Junior Hercilio⁶,Graner Edgard¹,Coletta Ricardo D.¹

Affiliation:

1. Department of Oral Diagnosis, School of DentistryUniversity of Campinas Piracicaba São Paulo Brazil

2. Molecular Biology DepartmentFederal University of Paraiba João Pessoa Paraiba Brazil

3. Department of Basic ScienceBahiana School of Medicine and Public Health Salvador Bahia Brazil

4. Department of Epidemiology and Biostatistics, School of Public HealthPeking University Beijing People's Republic of China

5. Department of Morphology, School of DentistryUniversity of Campinas Piracicaba São Paulo Brazil

6. Stomatology Clinic, Dental School, State University of Montes Claros, Montes Claros, Minas Gerais, Brazil and Center for Rehabilitation of Craniofacial AnomaliesUniversity of José Rosario Vellano Alfenas Minas Gerais Brazil

Funder

Fundação de Amparo à Pesquisa do Estado de São Paulo

Publisher

Wiley

Subject

Health, Toxicology and Mutagenesis,Genetics (clinical),Epidemiology

Link

https://onlinelibrary.wiley.com/doi/pdf/10.1002/em.22239

Reference42 articles.

1. A simple and cost-effective protocol for DNA isolation from buccal epithelial cells

2. Serum esterases. 2. An enzyme hydrolysing diethyl p-nitrophenyl phosphate (E 600) and its identity with the A-esterase of mammalian sera

3. Paraoxonases 1, 2, and 3, oxidative stress, and macrophage foam cell formation during atherosclerosis development