Increased EPO Levels Are Associated With Bone Loss in Mice Lacking PHD2 in EPO-Producing Cells-Reference-Cited by-同舟云学术

Increased EPO Levels Are Associated With Bone Loss in Mice Lacking PHD2 in EPO-Producing Cells

Published:2016-05-13 Issue:10 Volume:31 Page:1877-1887
ISSN:0884-0431
Container-title:Journal of Bone and Mineral Research
language:en
Short-container-title:J Bone Miner Res

Author:

Rauner Martina¹,Franke Kristin²³,Murray Marta²³,Singh Rashim Pal²³,Hiram-Bab Sahar⁴,Platzbecker Uwe⁵,Gassmann Max⁶⁷⁸,Socolovsky Merav⁹¹⁰,Neumann Drorit¹¹,Gabet Yankel⁴,Chavakis Triantafyllos²³¹²,Hofbauer Lorenz C¹¹²,Wielockx Ben²³¹²

Affiliation:

1. Department of Medicine III; Technische Universität Dresden; Dresden Germany

2. Department of Clinical Pathobiochemistry; Technische Universität Dresden; Dresden Germany

3. Institute for Clinical Chemistry and Laboratory Medicine; Technische Universität Dresden; Dresden Germany

4. Department of Anatomy and Anthropology; Sackler Faculty of Medicine; Tel-Aviv University; Tel Aviv Israel

5. Department of Medicine I; Technische Universität Dresden; Dresden Germany

6. Institute of Veterinary Physiology; Vetsuisse Faculty; University of Zürich; Zürich Switzerland

7. Zurich Center for Integrative Human Physiology (ZIHP); University of Zürich; Zürich Switzerland

8. Universidad Peruana Cayetano Heredia (UPCH); Lima Peru

9. Department of Molecular, Cell and Cancer Biology; University of Massachusetts Medical School; Worcester MA

10. Department of Pediatrics; University of Massachusetts Medical School; Worcester MA

11. Department of Cell and Developmental Biology; Sackler Faculty of Medicine; Tel-Aviv University; Tel Aviv Israel

12. Center for Regenerative Therapies Dresden; Dresden Germany

Funder

Deutsche Forschungsgemeinschaft

Publisher

Wiley

Subject

Orthopedics and Sports Medicine,Endocrinology, Diabetes and Metabolism

Reference42 articles.

1. Signaling pathways affecting skeletal health;Marie;Curr Osteoporos Rep,2012

2. Osteoblast recruitment to sites of bone formation in skeletal development, homeostasis, and regeneration;Dirckx;Birth Defects Res C Embryo Today,2013

3. Osteoblasts in osteoporosis: past, emerging, and future anabolic targets;Marie;Eur J Endocrinol,2011

4. Osteoporosis: now and the future;Rachner;Lancet,2011

5. HIF1alpha is a central regulator of collagen hydroxylation and secretion under hypoxia during bone development;Bentovim;Development,2012

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