Increased red cell turnover in a line of CD22-deficient mice is caused byGpi1c: A model for hereditary haemolytic anaemia-Reference-Cited by-同舟云学术

Increased red cell turnover in a line of CD22-deficient mice is caused byGpi1c: A model for hereditary haemolytic anaemia

Published:2012-10-16 Issue:12 Volume:42 Page:3212-3222
ISSN:0014-2980
Container-title:European Journal of Immunology
language:en
Short-container-title:Eur. J. Immunol.

Author:

Walker Jennifer A.¹,Hall Andrew M.²,Kotsopoulou Ekaterini³,Espeli Marion¹,Nitschke Lars⁴,Barker Robert N.²,Lyons Paul A.¹,Smith Kenneth G.C.¹

Affiliation:

1. Cambridge Institute for Medical Research and Department of Medicine; University of Cambridge School of Clinical Medicine; Addenbrooke's Hospital; Cambridge UK

2. Section of Immunology and Infection; Division of Applied Medicine; University of Aberdeen; Institute of Medical Sciences; Aberdeen UK

3. Cambridge Institute for Medical Research and Department of Haematology; University of Cambridge School of Clinical Medicine; Addenbrooke's Hospital; Cambridge UK

4. Department of Genetics; University of Erlangen; Erlangen Germany

Funder

Wellcome Trust PhD scholarship

Wellcome Trust Programme

KGCS is a Lister Prize Fellow

NIHR Cambridge Biomedical Research Centre

Cambridge Institute for Medical Research is in receipt of a Wellcome Trust Strategic Award

Publisher

Wiley

Subject

Immunology,Immunology and Allergy

Reference55 articles.