Intra-islet proliferation of cytotoxic T lymphocytes contributes to insulitis progression-Reference-Cited by-同舟云学术

Intra-islet proliferation of cytotoxic T lymphocytes contributes to insulitis progression

Published:2012-06-12 Issue:7 Volume:42 Page:1717-1722
ISSN:0014-2980
Container-title:European Journal of Immunology
language:en
Short-container-title:Eur. J. Immunol.

Author:

Graham Kate L.¹,Krishnamurthy Balasubramanian¹,Fynch Stacey¹,Ayala-Perez Rochelle¹,Slattery Robyn M.²,Santamaria Pere³,Thomas Helen E.,Kay Thomas W. H.

Affiliation:

1. St. Vincent's Institute; Fitzroy; Victoria; Australia

2. Department of Immunology; Faculty of Medicine; Nursing and Health Sciences; Monash University; The Alfred Hospital; Victoria; Australia

3. Julia McFarlane Diabetes Research Centre and Department of Microbiology; Immunology and Infectious Disease; Faculty of Medicine, University of Calgary; Calgary; Alberta; Canada

Publisher

Wiley

Subject

Immunology,Immunology and Allergy

Link

http://onlinelibrary.wiley.com/wol1/doi/10.1002/eji.201242435/fullpdf

Reference18 articles.

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2. Evolution of ectopic lymphoid neogenesis and in situ autoantibody production in autoimmune nonobese diabetic mice: cellular and molecular characterization of tertiary lymphoid structures in pancreatic islets;Astorri;J. Immunol.,2010

3. Prevention of diabetes by FTY720-mediated stabilization of peri-islet tertiary lymphoid organs;Penaranda;Diabetes,2010

4. FTY720: sphingosine 1-phosphate receptor-1 in the control of lymphocyte egress and endothelial barrier function;Brinkmann;Am. J. Transplant,2004

5. Autoreactive cytotoxic T lymphocytes acquire higher expression of cytotoxic effector markers in the islets of NOD mice after priming in pancreatic lymph nodes;Graham;Am. J. Pathol.,2011

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