<scp>CuATSM</scp> effectively ameliorates <scp>ALS</scp> patient astrocyte‐mediated motor neuron toxicity in human in vitro models of amyotrophic lateral sclerosis-Reference-Cited by-同舟云学术

CuATSM effectively ameliorates ALS patient astrocyte‐mediated motor neuron toxicity in human in vitro models of amyotrophic lateral sclerosis

Published:2022-10-10 Issue:2 Volume:71 Page:350-365
ISSN:0894-1491
Container-title:Glia
language:en
Short-container-title:Glia

Author:

Dennys Cassandra N.¹,Roussel Florence¹,Rodrigo Rochelle¹^ORCID,Zhang Xiaojin¹,Sierra Delgado Andrea¹,Hartlaub Annalisa¹,Saelim‐Ector Asya¹,Ray Will²,Heintzman Sarah³,Fox Ashley³,Kolb Stephen J.³⁴⁵⁶,Beckman Joseph⁷,Franco Maria Clara⁷,Meyer Kathrin¹⁵⁸^ORCID

Affiliation:

1. Center for Gene Therapy The Abigail Wexner Research Institute at Nationwide Children's Hospital Columbus Ohio USA

2. Mathematics Department The Abigail Wexner Research Institute at Nationwide Children's Hospital Columbus Ohio USA

3. Department of Neurology The Ohio State University Medical Center Columbus Ohio USA

4. Department of Biological Chemistry & Pharmacology The Ohio State University Wexner Medical Center Columbus Ohio USA

5. Molecular, Cellular & Developmental Biology Graduate Program The Ohio State University Columbus Ohio USA

6. Department of Neuroscience The Ohio State University Wexner Medical Center Columbus Ohio USA

7. Department of Biochemistry and Biophysics Oregon State University Corvallis Oregon USA

8. Department of Pediatrics The Ohio State University Medical Center Columbus Ohio USA

Publisher

Wiley

Subject

Cellular and Molecular Neuroscience,Neurology

Link

https://onlinelibrary.wiley.com/doi/pdf/10.1002/glia.24278

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2. Confirmatory double-blind, parallel-group, placebo-controlled study of efficacy and safety of edaravone (MCI-186) in amyotrophic lateral sclerosis patients

3. Upregulation of protein-tyrosine nitration in the anterior horn cells of amyotrophic lateral sclerosis

4. Prolactin response as an index of dopaminergic receptor function in Parkinson's disease. Correlation with clinical findings and therapeutic response

5. Astrocyte adenosine deaminase loss increases motor neuron toxicity in amyotrophic lateral sclerosis

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