Specific knockout of Sox2 in astrocytes reduces reactive astrocyte formation and promotes recovery after early postnatal traumatic brain injury in mouse cortex-Reference-Cited by-同舟云学术

Specific knockout of Sox2 in astrocytes reduces reactive astrocyte formation and promotes recovery after early postnatal traumatic brain injury in mouse cortex

Published:2022-11-10 Issue:3 Volume:71 Page:602-615
ISSN:0894-1491
Container-title:Glia
language:en
Short-container-title:Glia

Author:

Liu Yitong¹,Hong Wentong¹,Gong Pifang¹,Qi Guibo¹,Wang Xiaoxuan¹,Kang Siying¹,Tang Han¹,Qin Song¹²^ORCID

Affiliation:

1. Department of Anatomy, Histology and Embryology, School of Basic Medical Sciences Fudan University Shanghai China

2. State Key Laboratory of Medical Neurobiology and MOE Frontiers Center for Brain Science Fudan University Shanghai China

Funder

National Natural Science Foundation of China

Natural Science Foundation of Shanghai

National Key Research and Development Program of China

Publisher

Wiley

Subject

Cellular and Molecular Neuroscience,Neurology

Link

https://onlinelibrary.wiley.com/doi/pdf/10.1002/glia.24298

Reference64 articles.

2. Astrocyte glutamate transport: Review of properties, regulation, and physiological functions

3. Multipotent cell lineages in early mouse development depend on SOX2 function

4. Sox2‐dependent maintenance of mouse oligodendroglioma involves the Sox2‐mediated downregulation of Cdkn2b, Ebf1, Zfp423, and Hey2

5. Cathelicidin‐related antimicrobial peptide promotes neuroinflammation through astrocyte–microglia communication in experimental autoimmune encephalomyelitis

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