Fibrosis, Vascular Activation, and Immune Abnormalities Resembling Systemic Sclerosis in Bleomycin‐Treated Fli‐1–Haploinsufficient Mice-Reference-Cited by-同舟云学术

Fibrosis, Vascular Activation, and Immune Abnormalities Resembling Systemic Sclerosis in Bleomycin‐Treated Fli‐1–Haploinsufficient Mice

Published:2015-01-28 Issue:2 Volume:67 Page:517-526
ISSN:2326-5191
Container-title:Arthritis & Rheumatology
language:en
Short-container-title:Arthritis & Rheumatology

Author:

Taniguchi Takashi¹,Asano Yoshihide¹,Akamata Kaname¹,Noda Shinji¹,Takahashi Takehiro¹,Ichimura Yohei¹,Toyama Tetsuo¹,Trojanowska Maria²,Sato Shinichi¹

Affiliation:

1. University of Tokyo Graduate School of MedicineTokyoJapan

2. Boston University School of MedicineBostonMassachusetts

Funder

This work was supported by a grant for Research on Intractable Diseases from the Ministry of Health, Labour, and Welfare of Japan, from Japan Intractable Diseases Research Foundation, from Rohto Dermatology Prize, from JSID's Fellowship Shiseido Award, from the Mochida Memorial Foundation for Medica

Publisher

Wiley

Subject

Immunology,Rheumatology,Immunology and Allergy

Link

https://onlinelibrary.wiley.com/doi/pdf/10.1002/art.38948

Reference48 articles.

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4. Transforming Growth Factor-β Regulates DNA Binding Activity of Transcription Factor Fli1 by p300/CREB-binding Protein-associated Factor-dependent Acetylation

5. Interferon-γ promotes vascular remodeling in human microvascular endothelial cells by upregulating endothelin (ET)-1 and transforming growth factor (TGF) β2

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