Modular PROTAC Design for the Degradation of Oncogenic BCR-ABL
Author:
Funder
NIH
EMBO
Publisher
Wiley
Subject
General Chemistry,Catalysis
Reference22 articles.
1. Regulation of the c-Abl and Bcr–Abl tyrosine kinases
2. BCR-ABL uncouples canonical JAK2-STAT5 signaling in chronic myeloid leukemia
3. Molecular biology of bcr-abl1–positive chronic myeloid leukemia
4. Effects of a selective inhibitor of the Abl tyrosine kinase on the growth of Bcr–Abl positive cells
5. A novel mechanism for imatinib mesylate–induced cell death of BCR-ABL–positive human leukemic cells: caspase-independent, necrosis-like programmed cell death mediated by serine protease activity
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