Physiological aging at striatal synapses
Author:
Affiliation:
1. Leonard Davis School of Gerontology University of Southern California Los Angeles California
Publisher
Wiley
Subject
Cellular and Molecular Neuroscience
Link
https://onlinelibrary.wiley.com/doi/pdf/10.1002/jnr.24484
Reference77 articles.
1. Decreased Striatal Dopamine Release Underlies Increased Expression of Long-Term Synaptic Potentiation at Corticostriatal Synapses 24 h after 3-Nitropropionic-Acid-Induced Chemical Hypoxia
2. Brief mitochondrial inhibition causes lasting changes in motor behavior and corticostriatal synaptic physiology in the Fischer 344 rat
3. Functional state of corticostriatal synapses determines their expression of short- and long-term plasticity
4. Corticostriatal Paired-Pulse Potentiation Produced by Voltage-Dependent Activation of NMDA Receptors and L-Type Ca2+ Channels
5. Pre- and postsynaptic contributions to age-related alterations in corticostriatal synaptic plasticity
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