Arginine vasotocin, an endogenous neuropeptide ofAplysia, suppresses the gill withdrawal reflex and reduces the evoked synaptic input to central gill motor neurons-Reference-Cited by-同舟云学术

Arginine vasotocin, an endogenous neuropeptide ofAplysia, suppresses the gill withdrawal reflex and reduces the evoked synaptic input to central gill motor neurons

Published:1981-11 Issue:6 Volume:12 Page:533-544
ISSN:0022-3034
Container-title:Journal of Neurobiology
language:en
Short-container-title:J. Neurobiol.

Author:

Thornhill J. A.,Lukowiak K.,Cooper K. E.,Veale W. L.,Edstrom J. P.

Publisher

Wiley

Subject

Cellular and Molecular Neuroscience,General Neuroscience

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4. Presynaptic Facilitation as a Mechanism for Behavioral Sensitization in Aplysia

5. Quantitative analysis of relative contribution of central and peripheral neurons to gill-withdrawal reflex in Aplysia californica

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1. Characterization of an Aplysia vasotocin signaling system and actions of posttranslational modifications and individual residues of the ligand on receptor activity;Frontiers in Pharmacology;2023-03-20

2. Comparative and Evolutionary Physiology of Vasopressin/ Oxytocin-Type Neuropeptide Signaling in Invertebrates;Frontiers in Endocrinology;2020-04-17

3. Localization of vasopressin-like immunoreactivity in the CNS ofAplysia californica;Journal of Neurocytology;1992-09

4. A vasotocin-like peptide in Aplysia kurodai ganglia: HPLC and RIA evidence for its identity with Lys-conopressin G;Peptides;1992-05

5. Conopressin G, a molluscan vasopressin-like peptide, alters gill behaviors in Aplysia;Canadian Journal of Physiology and Pharmacology;1992-02-01