Genetic control of the survival of murine trisomy 16 fetuses
Author:
Publisher
Wiley
Subject
Health, Toxicology and Mutagenesis,Developmental Biology,Toxicology,Embryology
Reference20 articles.
1. Genesis and systematization of cardiovascular anomalies and analysis of skeletal malformations in murine trisomy 16 and 19
2. (1984) The dominant lethal assay in the male mouse. In: Handbook of Mutagenicity Test Procedures. and eds. Elsevier, Amsterdam, pp. 471-483.
3. and (1985) Cytogenetics of pregnancy wastage. In: Advances in Human Genetics, Vol. 14. and eds. Plenum Press, New York, pp. 1-37.
4. and (1987) Cytogenetics of Mammalian Embryonic Development. New York: Clarendon Press, Oxford Science Publications.
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1. Cytogenetics;Genetics of the Mouse;2014-11-30
2. Duplication of the entire 22.9 Mb human chromosome 21 syntenic region on mouse chromosome 16 causes cardiovascular and gastrointestinal abnormalities;Human Molecular Genetics;2007-04-05
3. DSCR1 gene expression is dependent on NFATc1 during cardiac valve formation and colocalizes with anomalous organ development in trisomy 16 mice;Developmental Biology;2004-02
4. Effects of genetic background on cardiovascular anomalies in the Ts16 mouse;Developmental Dynamics;2004
5. Identification of Trisomy 16 Murine Embryos by Fluorescence In Situ Hybridization;BioTechniques;2001-08
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