Genetic independence of the embryonic reactivity difference to cortisone- and 6-aminonicotinamide-induced cleft palate in the mouse
Author:
Publisher
Wiley
Subject
Health, Toxicology and Mutagenesis,Developmental Biology,Toxicology,Embryology
Reference20 articles.
1. Teratogenesis of acetazolamide in the CBA/J and SWV strains of mice. II. Genetic control of the teratogenic response
2. 6-Aminonicotinamide-induced cleft palate in the mouse: The nature of the difference between the A/J and C57BL/6J strains in frequency of response and its genetic basis
3. Use of dose-response relationships to discriminate between the mechanisms of cleft-palate induction by different teratogens: An argument for discussion
4. GENETICS OF CORTISONE-INDUCED CLEFT PALATE IN THE MOUSE—EMBRYONIC AND MATERNAL EFFECTS
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1. Correlation of susceptibility to 6-aminonicotinamide and hydrocortisone-induced cleft palate;Life Sciences;2005-03
2. The degree of lateralization of paw usage (handedness) in the mouse is defined by three major phenotypes;Behavior Genetics;1996-07
3. Time-dependent differences in the development of somites of four different mouse strains;Teratogenesis, Carcinogenesis, and Mutagenesis;1993
4. Potentiation of chemically induced cleft palate by ethanol ingestion during gestation in the mouse;Teratogenesis, Carcinogenesis, and Mutagenesis;1985
5. Increased susceptibility to 6-aminonicotinamide-induced cleft lip of heterozygote Dancer mice;Teratology;1984-02
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