Tumour extracellular vesicle‐derived Complement Factor H promotes tumorigenesis and metastasis by inhibiting complement‐dependent cytotoxicity of tumour cells-Reference-Cited by-同舟云学术

Tumour extracellular vesicle‐derived Complement Factor H promotes tumorigenesis and metastasis by inhibiting complement‐dependent cytotoxicity of tumour cells

Published:2020-11 Issue:1 Volume:10 Page:
ISSN:2001-3078
Container-title:Journal of Extracellular Vesicles
language:en
Short-container-title:Journal of Extracellular Vesicles

Author:

Mao Xiaowen¹,Zhou Longyin¹,Tey Sze Keong¹,Ma Angel Po Yee¹,Yeung Cherlie Lot Sum¹,Ng Tung Him¹,Wong Samuel Wan Ki¹,Liu Bonnie Hei Man¹,Fung Yi Man Eva²,Patz Edward F.³⁴,Cao Peihua⁵⁶,Gao Yi⁶⁷⁸⁹,Yam Judy Wai Ping¹⁶¹⁰^ORCID

Affiliation:

1. Department of Pathology, Li Ka Shing Faculty of Medicine The University of Hong Kong Pokfulam Hong Kong

2. Department of Chemistry, State Key Laboratory of Synthetic Chemistry The University of Hong Kong Pokfulam Hong Kong

3. Department of Radiology Duke University Medical Center Durham USA

4. Department of Pharmacology and Cancer Biology Duke University Medical Center Durham USA

5. Clinical Research Center, Zhujiang Hospital Southern Medical University Guangzhou Guangdong P. R. China

6. Department of Hepatobiliary Surgery II, Zhujiang Hospital Southern Medical University Guangzhou Guangdong P. R. China

7. Institute of Regenerative Medicine, Zhujiang Hospital Southern Medical University Guangzhou P. R. China

8. Artificial Organs and Tissue Engineering Centre of Guangdong Province Guangzhou P. R. China

9. State Key Laboratory of Organ Failure Research Southern Medical University Guangzhou P. R. China

10. State Key Laboratory of Liver Research (The University of Hong Kong) Pokfulam Hong Kong

Publisher

Wiley

Subject

Cell Biology,Histology

Link

https://onlinelibrary.wiley.com/doi/pdf/10.1002/jev2.12031

Reference50 articles.

1. Down-Regulation of Human Complement Factor H Sensitizes Non-Small Cell Lung Cancer Cells to Complement Attack and Reduces In Vivo Tumor Growth

2. Intercellular transfer of the oncogenic receptor EGFRvIII by microvesicles derived from tumour cells

3. Complement Factor H Autoantibodies Are Associated with Early Stage NSCLC

4. Structural basis for sialic acid–mediated self-recognition by complement factor H

5. Complement Component 3 Adapts the Cerebrospinal Fluid for Leptomeningeal Metastasis

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