Heme oxygenase‐1 reduces the sensitivity to imatinib through nonselective activation of histone deacetylases in chronic myeloid leukemia-Reference-Cited by-同舟云学术

Heme oxygenase‐1 reduces the sensitivity to imatinib through nonselective activation of histone deacetylases in chronic myeloid leukemia

Published:2018-09-07 Issue:4 Volume:234 Page:5252-5263
ISSN:0021-9541
Container-title:Journal of Cellular Physiology
language:en
Short-container-title:J Cell Physiol

Author:

Wei Danna¹²³^ORCID,Lu Tingting¹²³,Ma Dan¹²,YU Kunlin¹²³,Li Xinyao¹²³,Chen Bingqing¹²,Xiong Ji¹²,Zhang Tianzhuo¹²³,Wang Jishi¹²³

Affiliation:

1. Department of Hematology Affiliated Hospital of Guizhou Medical University Guiyang PR China

2. Department of Key Laboratory of Hematological Disease Diagnostic and Treatment Centre Guizhou Province Hematopoietic Stem Cell Transplantation Center Guiyang PR China

3. Department of Clinical Medical School Guizhou Medical University Guiyang PR China

Funder

National Natural Science Foundation of China

Publisher

Wiley

Subject

Cell Biology,Clinical Biochemistry,Physiology

Link

https://onlinelibrary.wiley.com/doi/pdf/10.1002/jcp.27334

Reference45 articles.

1. A Redox-Dependent Pathway for Regulating Class II HDACs and Cardiac Hypertrophy

2. Bortezomib induces heme oxygenase-1 expression in multiple myeloma

3. Anticancer activities of histone deacetylase inhibitors

4. Heme oxygenase-1 contributes to imatinib resistance by promoting autophagy in chronic myeloid leukemia through disrupting the mTOR signaling pathway

5. Heme Oxygenase-1 Protects Retinal Endothelial Cells against High Glucose- and Oxidative/Nitrosative Stress-Induced Toxicity

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