Endogenous thymidine and hypoxanthine are a source of error in evaluating methotrexate cytotoxicity by clonogenic assays using undialyzed fetal bovine serum-Reference-Cited by-同舟云学术

Endogenous thymidine and hypoxanthine are a source of error in evaluating methotrexate cytotoxicity by clonogenic assays using undialyzed fetal bovine serum

Published:1986-01 Issue:1 Volume:4 Page:51-62
ISSN:0737-1454
Container-title:The International Journal of Cell Cloning
language:en
Short-container-title:The International Journal of Cell Cloning

Author:

Sobrero Alberto F.¹,Bertino Joseph R.¹

Affiliation:

1. Department of Pharmacology, Yale University School of Medicine, P.O. Box 3333, New Haven, CT, USA

Publisher

Wiley

Subject

Cell Biology

Link

https://onlinelibrary.wiley.com/doi/pdf/10.1002/stem.5530040106

Reference16 articles.

1. A Critical Appraisal of the Human Tumor Stem-Cell Assay

2. The reversal of methotrexate toxicity to mouse bone marrow cells by leucovorin and nucleosides;Pinedo HM;Cancer Res,1976

3. Resistance of human bone marrow CFU‐c to high‐dose methotrexate toxicity;Koizumi S;Exp Hematol,1980

4. Reversal of methotrexate inhibition of colony growth of L1210 leukemia cells in semisolid medium;Nederbragt H;Cancer Res,1981

5. The failure of methotrexate to inhibit chicken fibroblast proliferation in a serum containing culture medium;Mitchell RS;Cancer Res,1975

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