Mutant p53 in concert with an interleukin-27 receptor alpha deficiency causes spontaneous liver inflammation, fibrosis, and steatosis in mice
Author:
Affiliation:
1. Department of Pediatrics; The University of Texas MD Anderson Cancer Center; Houston TX
2. Department of Genetics; The University of Texas MD Anderson Cancer Center; Houston TX
Publisher
Wiley
Subject
Hepatology
Link
http://onlinelibrary.wiley.com/wol1/doi/10.1002/hep.28379/fullpdf
Reference36 articles.
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2. Gain of function of a p53 hot spot mutation in a mouse model of Li-Fraumeni syndrome;Lang;Cell,2004
3. Molecular mechanisms of oncogene-induced inflammation and inflammation-sustained oncogene activation in gastrointestinal tumors: an underappreciated symbiotic relationship;Dibra;Biochim Biophys Acta,2014
4. Mutant p53 prolongs NF-kappaB activation and promotes chronic inflammation and inflammation-associated colorectal cancer;Cooks;Cancer Cell,2013
5. Serum cytokine and soluble cytokine receptor levels in patients with non-alcoholic steatohepatitis;Abiru;Liver Int,2006
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