PNPLA3, CGI‐58, and Inhibition of Hepatic Triglyceride Hydrolysis in Mice-Reference-Cited by-同舟云学术

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PNPLA3, CGI‐58, and Inhibition of Hepatic Triglyceride Hydrolysis in Mice

Published:2019-04-09 Issue: Volume: Page:
ISSN:0270-9139
Container-title:Hepatology
language:en
Short-container-title:Hepatology

Author:

Wang Yang¹,Kory Nora¹,BasuRay Soumik¹,Cohen Jonathan C.²³,Hobbs Helen H.¹²⁴

Affiliation:

1. Department of Molecular Genetics University of Texas Southwestern Medical Center Dallas TX

2. Department of Internal Medicine University of Texas Southwestern Medical Center Dallas TX

3. The Center for Human Nutrition University of Texas Southwestern Medical Center Dallas TX

4. Howard Hughes Medical Institute University of Texas Southwestern Medical Center Dallas TX

Funder

National Institutes of Health

National Institute of Diabetes and Digestive and Kidney Diseases

Publisher

Wiley

Subject

Hepatology

Link

https://onlinelibrary.wiley.com/doi/pdf/10.1002/hep.30583

Reference37 articles.

1. Liver diseases: A major, neglected global public health problem requiring urgent actions and large-scale screening

2. Nonalcoholic fatty liver disease and hepatic cirrhosis: Comparison with viral hepatitis-associated steatosis

3. Nonalcoholic fatty liver disease: epidemiology, pathogenesis and therapeutic implications;Andronescu CI;J Med Life,2018

4. Genetic variation in PNPLA3 confers susceptibility to nonalcoholic fatty liver disease

5. Variant in PNPLA3 is associated with alcoholic liver disease

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5. Coix Sprouts Affect Triglyceride Metabolism in Huh7 Cells and High-Fat Diet-Induced Obese Mice;Journal of Medicinal Food;2024-08-01

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