Salidroside ameliorates pathological cardiac hypertrophy via TLR4‐TAK1 ‐dependent signaling
Author:
Affiliation:
1. Department of Cardiology Renmin Hospital of Wuhan University Wuhan People's Republic of China
2. Hubei Key Laboratory of Metabolic and Chronic Diseases Wuhan People's Republic of China
Funder
National Natural Science Foundation of China
Publisher
Wiley
Subject
Pharmacology
Link
https://onlinelibrary.wiley.com/doi/pdf/10.1002/ptr.7701
Reference47 articles.
1. Integrins Play a Critical Role in Mechanical Stress–Induced p38 MAPK Activation
2. Cardioprotective effects of salidroside on myocardial ischemia–reperfusion injury in coronary artery occlusion-induced rats and Langendorff-perfused rat hearts
3. Thaliporphine ameliorates cardiac depression in endotoxemic rats through attenuating TLR4 signaling in the downstream of TAK-1 phosphorylation and NF-κB signaling
4. Regulation of cardiac hypertrophy in vivo by the stress-activated protein kinases/c-Jun NH2-terminal kinases
5. Central blockade of TLR4 improves cardiac function and attenuates myocardial inflammation in angiotensin II-induced hypertension
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