Downregulation of the Ca 2+ ‐activated K + channel K C a 3.1 by histone deacetylase inhibition in human breast cancer cells-Reference-Cited by-同舟云学术

Downregulation of the Ca 2+ ‐activated K + channel K C a 3.1 by histone deacetylase inhibition in human breast cancer cells

Published:2016-03-17 Issue:2 Volume:4 Page:
ISSN:2052-1707
Container-title:Pharmacology Research & Perspectives
language:en
Short-container-title:Pharmacol Res Perspect

Author:

Ohya Susumu¹,Kanatsuka Saki¹,Hatano Noriyuki²,Kito Hiroaki¹,Matsui Azusa¹,Fujimoto Mayu¹,Matsuba Sayo¹,Niwa Satomi¹,Zhan Peng³,Suzuki Takayoshi³,Muraki Katsuhiko²

Affiliation:

1. Department of Pharmacology Division of Pathological Sciences Kyoto Pharmaceutical University Kyoto 607‐8414 Japan

2. Laboratory of Cellular Pharmacology School of Pharmacy Aichi‐Gakuin University Nagoya 464‐8650 Japan

3. Graduate School of Medical Science Kyoto Prefectural University of Medicine Kyoto 606‐0823 Japan

Funder

Japan Society for the Promotion of Science

Mochida Memorial Foundation for Medical and Pharmaceutical Research

Uehara Memorial Foundation

Publisher

Wiley

Subject

General Pharmacology, Toxicology and Pharmaceutics,Neurology

Link

https://onlinelibrary.wiley.com/doi/pdf/10.1002/prp2.228

Reference56 articles.

1. Identification of Differentially Expressed IGFBP5-Related Genes in Breast Cancer Tumor Tissues Using cDNA Microarray Experiments

2. Nucleoside diphosphate kinase as protein histidine kinase

3. Emerging roles of store-operated Ca2+entry through STIM and ORAI proteins in immunity, hemostasis and cancer

4. Histone deacetylase inhibitor (HDACI) mechanisms of action: Emerging insights

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