CD16‐158‐valine chimeric receptor T cells overcome the resistance of KRAS‐mutated colorectal carcinoma cells to cetuximab-Reference-Cited by-同舟云学术

CD16‐158‐valine chimeric receptor T cells overcome the resistance of KRAS‐mutated colorectal carcinoma cells to cetuximab

Published:2019-08-30 Issue:9 Volume:146 Page:2531-2538
ISSN:0020-7136
Container-title:International Journal of Cancer
language:en
Short-container-title:Int. J. Cancer

Author:

Arriga Roberto¹,Caratelli Sara²,Lanzilli Giulia²,Ottaviani Alessio²,Cenciarelli Carlo²,Sconocchia Tommaso³,Spagnoli Giulio C.²,Iezzi Giandomenica⁴,Roselli Mario¹,Lauro Davide¹,Coppola Andrea¹,Dotti Gianpietro⁵,Ferrone Soldano⁶,Sconocchia Giuseppe²^ORCID

Affiliation:

1. Department of Systems Medicine, Endocrinology and Medical Oncology University of Rome “Tor Vergata” Rome Italy

2. Institute of Translational Pharmacology CNR Rome Italy

3. Otto Loewi Research Center Chair of Immunology and Pathophysiology Medical University of Graz Graz Austria

4. Department of Surgery, Ente Ospedaliero Cantonale Università della Svizzera Italiana Lugano Switzerland

5. Lineberger Comprehensive Cancer Center University of North Carolina Chapel Hill North Carolina

6. Department of Surgery, Massachusetts General Hospital Harvard Medical School Boston Massachusetts

Funder

Associazione Italiana per la Ricerca sul Cancro

Foundation for the National Institutes of Health

Publisher

Wiley

Subject

Cancer Research,Oncology

Link

https://onlinelibrary.wiley.com/doi/pdf/10.1002/ijc.32618

Reference29 articles.

1. EGFR Targeting of Solid Tumors

2. Anti-epidermal growth factor receptor monoclonal antibodies in cancer therapy

3. K-rasMutations and Benefit from Cetuximab in Advanced Colorectal Cancer

4. Panitumumab–FOLFOX4 Treatment and RAS Mutations in Colorectal Cancer

5. Combination of NK Cells and Cetuximab to Enhance Anti-Tumor Responses in RAS Mutant Metastatic Colorectal Cancer

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