Lubricin Restoration in a Mouse Model of Congenital Deficiency-Reference-Cited by-同舟云学术

Lubricin Restoration in a Mouse Model of Congenital Deficiency

Published:2015-10-28 Issue:11 Volume:67 Page:3070-3081
ISSN:2326-5191
Container-title:Arthritis & Rheumatology
language:en
Short-container-title:Arthritis & Rheumatology

Author:

Hill Adele¹,Waller Kimberly A.²,Cui Yajun³,Allen Justin M.¹,Smits Patrick³,Zhang Ling X.²,Ayturk Ugur M.¹,Hann Steven³,Lessard Samantha G.³,Zurakowski David³,Warman Matthew L.⁴,Jay Gregory D.⁵

Affiliation:

1. Boston Children's Hospital and Harvard Medical School; Boston Massachusetts

2. Alpert Medical School of Brown University and Rhode Island Hospital; Providence

3. Boston Children's Hospital; Boston Massachusetts

4. Howard Hughes Medical Institute, Boston Children's Hospital, and Harvard Medical School; Boston Massachusetts

5. Rhode Island Hospital, Alpert Medical School of Brown University, and Brown University School of Engineering; Providence Rhode Island

Funder

NIH

Publisher

Wiley

Subject

Immunology,Rheumatology,Immunology and Allergy

Reference47 articles.

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2. CACP, encoding a secreted proteoglycan, is mutated in camptodactyly-arthropathy-coxa vara-pericarditis syndrome;Marcelino;Nat Genet,1999

3. Lubricin is a product of megakaryocyte stimulating factor gene expression by human synovial fibroblasts;Jay;J Rheumatol,2000

4. The secreted glycoprotein lubricin protects cartilage surfaces and inhibits synovial cell overgrowth;Rhee;J Clin Invest,2005

5. Immunodetection and partial cDNA sequence of the proteoglycan, superficial zone protein, synthesized by cells lining synovial joints;Schumacher;J Orthop Res,1999

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