Heat-shock protein-90 prolongs septic neutrophil survival by protecting c-Src kinase and caspase-8 from proteasomal degradation-Reference-Cited by-同舟云学术

Heat-shock protein-90 prolongs septic neutrophil survival by protecting c-Src kinase and caspase-8 from proteasomal degradation

Published:2018-02-02 Issue:5 Volume:103 Page:933-944
ISSN:0741-5400
Container-title:Journal of Leukocyte Biology
language:en
Short-container-title:J Leukoc Biol

Author:

Gupta Sahil¹²³,Lee Chan-Mi⁴⁵,Wang Jia-Feng⁶,Parodo Jean¹²,Jia Song-Hui¹²,Hu Jim⁴⁵,Marshall John C.¹²³

Affiliation:

1. The Keenan Research Centre for Biomedical Science; Li Ka Shing Knowledge Institute, St. Michael's Hospital; Toronto Ontario Canada

2. Departments of Surgery and Critical Care Medicine; St. Michael's Hospital, University of Toronto; Toronto Ontario Canada

3. Institute of Medical Science, Faculty of Medicine; University of Toronto; Toronto Ontario Canada

4. Department of Laboratory Medicine and Pathobiology; University of Toronto; Toronto Ontario Canada

5. Hospital for Sick Children Research Institute, Program in Physiology and Experimental Medicine, Peter Gilgan Centre for Research and Learning, Hospital for Sick Children; Toronto Ontario Canada

6. Department of Anesthesiology and Intensive Care; Changhai Hospital, The Second Military Medical University; Shanghai China

Funder

Canadian Institutes of Health Research

Physicians' Services Incorporated Foundation

Publisher

Wiley

Subject

Cell Biology,Immunology,Immunology and Allergy

Reference47 articles.

1. Assessment of global incidence and mortality of hospital-treated sepsis-current estimates and limitations.;Fleischmann;Am J Respir Critic Care Med,2016

2. Pre-B cell colony-enhancing factor inhibits neutrophil apoptosis in experimental inflammation and clinical sepsis;Jia SH;J Clin Invest,2004

3. HSP90 inhibition suppresses lipopolysaccharide-induced lung inflammation in vivo;Lilja;PLoS One,2015

4. Inhibitory effects of heat shock protein 90 blockade on proinflammatory human Th1 and Th17 cell subpopulations;Tukaj;J Inflamm (Lond),2014

5. Why have clinical trials in sepsis failed?;Marshall;Trends Mol Med,2014

Cited by 24 articles. 订阅此论文施引文献订阅此论文施引文献，注册后可以免费订阅5篇论文的施引文献，订阅后可以查看论文全部施引文献

1. MYOCD and SRF-mediated MLCK transcription prevents polymorphonuclear neutrophils from ferroptosis in sepsis-related acute lung injury;Immunologic Research;2024-08-28

2. Cerebral Endothelial CXCR2 Promotes Neutrophil Transmigration into Central Nervous System in LPS-Induced Septic Encephalopathy;Biomedicines;2024-07-11

3. Prediction of Prognosis in Patients with Sepsis Based on Platelet-Related Genes;Hormone and Metabolic Research;2024-06-13

4. Reframing sepsis immunobiology for translation: towards informative subtyping and targeted immunomodulatory therapies;The Lancet Respiratory Medicine;2024-04

5. Preventing occludin tight-junction disruption via inhibition of microRNA-193b-5p attenuates viral load and influenza-induced lung injury;Molecular Therapy;2023-09