The 3‐NP Model of Striatal Neurodegeneration
Author:
Affiliation:
1. Neurodegenerative Diseases Laboratory, URA2210, CEA, and CNRS, Molecular Imaging Research Center (MIRCen), I2BM, Life Science Division Fontenay‐aux‐Roses France
Publisher
Wiley
Subject
General Medicine
Link
https://onlinelibrary.wiley.com/doi/pdf/10.1002/0471142301.ns0948s67
Reference31 articles.
1. Mitochondria take center stage in aging and neurodegeneration
2. Neurochemical and histologic characterization of striatal excitotoxic lesions produced by the mitochondrial toxin 3-nitropropionic acid
3. Involvement of Mitochondrial Complex II Defects in Neuronal Death Produced by N-Terminus Fragment of Mutated Huntingtin
4. Calpain Is a Major Cell Death Effector in Selective Striatal Degeneration InducedIn Vivoby 3-Nitropropionate: Implications for Huntington's Disease
5. A Dual Role of Adenosine A2AReceptors in 3-Nitropropionic Acid-Induced Striatal Lesions: Implications for the Neuroprotective Potential of A2AAntagonists
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